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Mitochondrial-Derived Peptide
MOTS-c is a mitochondrial-derived peptide studied in laboratory research for its involvement in cellular energy regulation and metabolic signaling pathways. Research often focuses on its interaction with mitochondrial function, stress-response mechanisms, and pathways related to cellular homeostasis in controlled experimental models.
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This product is intended strictly for laboratory research use within the United States. It is not approved by the FDA for the diagnosis, treatment, cure, or prevention of any disease. Not for human or veterinary use.
By purchasing, you confirm the material will be used solely for lawful research purposes in accordance with applicable U.S. federal, state, and local regulations.
MOTS-c (Mitochondrial Open Reading Frame of the 12S rRNA-c) is a mitochondrial-derived peptide identified through genomic analysis of mitochondrial DNA. It is synthesized for controlled laboratory research and studied within experimental environments focused on cellular and metabolic signaling.
In scientific literature, MOTS-c is examined for its interaction with metabolic regulation pathways, mitochondrial signaling mechanisms, and stress-response models under in vitro and preclinical research conditions.
MOTS-c is a 16-amino acid peptide encoded within mitochondrial DNA and identified in 2015 as part of the expanding class of mitochondrial-derived peptides. Unlike nuclear-encoded peptides, MOTS-c originates from the mitochondrial genome and has been studied for its role in intracellular communication and metabolic pathway regulation.
Research has examined its interaction with AMPK-related pathways, cellular stress response systems, and metabolic signaling frameworks under controlled experimental conditions.
MOTS-c was first characterized in 2015 during investigations into small open reading frames encoded by mitochondrial DNA. Its discovery contributed to the recognition of mitochondria as signaling organelles beyond their traditional bioenergetic role.
Subsequent research expanded into metabolic regulation and cellular stress-response models within preclinical laboratory studies.
Laboratory investigations into MOTS-c have explored its involvement in mitochondrial signaling and metabolic regulation pathways. Research models examine intracellular communication mechanisms, AMPK-associated signaling, and stress-response modulation under controlled in vitro and preclinical conditions.
No. Eppix Labs products are supplied exclusively for laboratory research. We do not provide dosing, administration, or usage guidance.
Each vial contains the labeled quantity (10mg or 45mg) of lyophilized MOTS-c. Independent third-party testing verifies purity, identity, and net content per batch.
The vial contains only the lyophilized compound. Any laboratory materials required for reconstitution or experimental procedures must be sourced separately.
Duration depends entirely on research protocol, storage conditions, and laboratory application.
The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance.
PubMedMOTS-c is an exercise-induced mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis.
PubMedNaturally occurring mitochondrial-derived peptides are age-dependent regulators of apoptosis, insulin sensitivity, and inflammatory markers. Aging (Albany NY) 8(4):796–809.
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