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Nootropic Peptide
Also known as Adamantane-modified Semax analogue
Adamax is a synthetic peptide supplied for laboratory research, described by its originators as an adamantane-modified analogue of the ACTH(4-10) fragment peptide Semax.
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This product is intended strictly for laboratory research use within Canada. It is not approved by Health Canada for the diagnosis, treatment, cure, or prevention of any disease. Not for human or veterinary use.
By purchasing, you confirm the material will be used solely for lawful research purposes in accordance with applicable Canadian regulations.
Adamax is a synthetic peptide supplied for laboratory research, described by its originators as an adamantane-modified analogue of the ACTH(4-10) fragment peptide Semax.
No primary study of Adamax itself has been published. The experimental framing below describes the Semax scaffold it is derived from, not Adamax.
Third-party tested for purity, ID, quantity.
Adamax is offered in the research market as a structural modification of N-acetyl Semax, with an adamantane group intended to alter stability and lipophilicity relative to the parent heptapeptide. The parent compound, Semax, is a synthetic analogue of the ACTH(4-10) fragment that has been characterized in Russian and international neuropharmacology research for effects on neurotrophin gene expression.
Because the adamantane-modified form has not itself been described in the peer-reviewed literature, its stability, receptor behaviour, and activity remain uncharacterized. Researchers should treat published Semax data as background on the scaffold rather than as evidence about Adamax.
Semax was developed from research on short ACTH fragments lacking hormonal activity, and has been studied for neurotrophic and cognitive endpoints since the 1980s. Structural work on the N-terminal region established that modification changes the expression of its nootropic profile.
Adamax emerged subsequently as a research-market modification of that scaffold. It has no published synthesis, characterization, or activity study of its own.
There is no compound-specific literature for Adamax. The references below characterize Semax, the ACTH(4-10) analogue scaffold from which Adamax is described as being derived, and cover neurotrophin gene expression, receptor binding, and behavioural endpoints in rodent models.
No. Eppix Labs products are supplied exclusively for laboratory research. We do not provide dosing, administration, or usage guidance.
Each unit contains the labeled quantity of Adamax. Independent third-party analysis verifies purity, identity, and net content per batch.
The unit contains only the research compound. Any laboratory materials required for reconstitution or experimental procedures must be sourced separately.
Duration depends entirely on research design, storage conditions, and laboratory protocol.
Semax, an analog of ACTH(4-10) with cognitive effects, regulates BDNF and trkB expression in the rat hippocampus
PubMedSemax, synthetic ACTH(4-10) analogue, attenuates behavioural and neurochemical alterations following early-life fluvoxamine exposure in white rats
PubMedNeurotrophin gene expression in rat brain under the action of Semax, an analogue of ACTH 4-10
PubMedEffect of modification of the N-terminal region of molecule on the expression of neotropic effect of semax analogues
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